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dc.contributor.authorAksara Thongprachumen_US
dc.contributor.authorShoko Okitsuen_US
dc.contributor.authorPattara Khamrinen_US
dc.contributor.authorNiwat Maneekarnen_US
dc.contributor.authorSatoshi Hayakawaen_US
dc.contributor.authorHiroshi Ushijimaen_US
dc.date.accessioned2018-09-05T03:26:54Z-
dc.date.available2018-09-05T03:26:54Z-
dc.date.issued2017-07-01en_US
dc.identifier.issn15677257en_US
dc.identifier.issn15671348en_US
dc.identifier.other2-s2.0-85016044108en_US
dc.identifier.other10.1016/j.meegid.2017.03.020en_US
dc.identifier.urihttps://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85016044108&origin=inwarden_US
dc.identifier.urihttp://cmuir.cmu.ac.th/jspui/handle/6653943832/56494-
dc.description.abstract© 2017 Elsevier B.V. In mid-2016, norovirus GII.2 emerged as a major cause of gastroenteritis outbreak in Japan with overall detection rate of 56.3% of norovirus cases. The differences in ORF1 and ORF2 of some norovirus GII were observed. Inter-subtype recombinants GII.Pe/GII.2, GII.P16/GII.2 and GII.P17/GII.2 were detected. Three amino acid substitutions were noted at P2 antigenic site of GII.P16/GII.2 recombinants. Furthermore, this study revealed that the current immunochromatographic kit available in Japan could be used effectively for the detection of recent GII.2 genotype.en_US
dc.subjectAgricultural and Biological Sciencesen_US
dc.subjectBiochemistry, Genetics and Molecular Biologyen_US
dc.subjectImmunology and Microbiologyen_US
dc.subjectMedicineen_US
dc.titleEmergence of norovirus GII.2 and its novel recombination during the gastroenteritis outbreak in Japanese children in mid-2016en_US
dc.typeJournalen_US
article.title.sourcetitleInfection, Genetics and Evolutionen_US
article.volume51en_US
article.stream.affiliationsNihon University School of Medicineen_US
article.stream.affiliationsUniversity of Tokyoen_US
article.stream.affiliationsChiang Mai Universityen_US
Appears in Collections:CMUL: Journal Articles

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